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We showed that i Gab1 is specifically expressed in OLs and oppositely regulated by triiodothyronine and PDGF; ii Gab1 is regulated by PDGF but not other growth factors in OLs; iii Gab1 deletion in OLs causes hypomyelination in the CNS by reducing OPC differentiation; iv Gab1 binds to GSK3ОІ and regulates its activity; and v Gab1 affects nuclear accumulation of ОІ catenin and regulates the expression of a number of factors critical to the transcription of myelin proteins <a href=http://vlasixs.cyou>lasix purpose</a> All recommendations refer to patients who present with a hormone receptor positive, HER2 not overexpressed, axillary node negative early breast cancer